Dead Arm Syndrome: Difference between revisions

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== Clinically Relevant Anatomy<br>  ==


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== Mechanism of Injury / Pathological Process<br>  ==
 
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== Clinical Presentation  ==
 
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== Diagnostic Procedures  ==
 
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== Outcome Measures  ==
 
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== Management / Interventions<br>  ==
 
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== Differential Diagnosis<br>  ==
 
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== Key Evidence  ==
 
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== References ==
== Definition/Description<br> ==


References will automatically be added here, see [[Adding References|adding references tutorial]].  

“Dead Arm” is characterized by a sudden sharp or ’paralyzing’ pain when the shoulder is moved forcibly into a position of maximum external rotation in elevation or is subjected to a direct blow. The patient is no longer able to perform a throwing movement with the control and the velocity that he achieved before the injury due to pain and numbness. It’s also called recurrent transient subluxation of the shoulder.<ref name="6a">CR Rowe and B Zarins, Recurrent transient subluxation of the shoulder, J Bone Joint Surg Am. 1981;63:863-872. level of evidence: 2B</ref><ref name="8">http://www.coreconcepts.com.sg/mcr/the-disabled-throwing-shoulder-the-“dead-arm”/</ref>&nbsp;
The dead arm syndrome is typically associated with anterior instability and a damaged anterior labrum, probably as a result of subluxation of the humeral head. This causes a transient stretch to the brachial plexus during a hard throw.<ref name="10">Richard B. Birrer,Bernard A.. Griesemer,Mary B. Cataletto, M.D.Pediatric sports medicine for primary care, 2002,  p348</ref>&nbsp;
The phenomenon is a disorder that can have different causes. Mostly it are problems of the rotator cuff or the labrum. Instability of the shoulder or posterior capsular contracture may be a reason for the development of the dead arm syndrome. In addition, it can also be caused by calcification in the ball and socket joint, bone spurs in the acromion, impingement of the shoulder ligaments, biceps tendonitis, micro-instability, internal impingement and SLAP lesion. Psychological factors can also cause this condition. This syndrome may also occur during throwing, repetitive forceful serving in tennis, or working with the arm in a strained position above shoulder.<ref name="6a" /><ref name="8" /><ref name="10" />&nbsp; 
The symptoms can exacerbate by the loss of the posterior rollback. This leads to anterior translation and results in greater internal impingement posteriorly.<ref name="12">Donald H. Johnson, M.D, Practical orthopaedic sports medicine &amp;amp;amp; arthroscopy, 2007</ref>&nbsp;


<references />.  
<br>There are 2 categories of the dead arm syndrome: aware or unaware of subluxation.  


<br>  
<br>  


Dead arm syndrome<br>Contents<br>1 Search strategy<br>2.Definition/ Description<br>3 Clinically Relevant Anatomy<br>4 Epidemiology /Etiology<br>5 Differential Diagnosis<br>6 Examination<br>7 Medical Management<br>8 Physical Therapy Management<br>9 Key Research<br>10 Resources
<br>  


1. Search strategy<br>Database: Pubmed, Web of knowledge<br>Keywords: dead arm syndrome, recurrent transient subluxation of the shoulder, dead arm syndrome AND physiotherapy
== Epidemiology / Etiology<br> ==


2. Definition/ description = <br>  
The dead arm syndrome is seen most commonly in young athletes (21-30 years) or individuals whose arms have been powerful hyperextended in elevation and external rotation of the shoulder. <ref name="6a" /> &nbsp;<br>It’s common for people who participate in repetitive throwing sports because the arm is repetitive being turned out backwards as far as possible (external rotation) to create potential energy in the wind up phase prior to the forward acceleration phase. It’s been postulated that the pain is due to an overstretched anterior capsule of the shoulder. Because of this, the ball of the shoulder can shift forward creating an impingement of structures lying in the front of the shoulder joint, which result in pain an the inability to throw. <ref name="8" /> &nbsp;


“Dead Arm” is characterized by a sudden sharp or ’paralyzing’ pain when the shoulder is moved forcibly into a position of maximum external rotation in elevation or is subjected to a direct blow. The patient is no longer able to perform a throwing movement with the control and the velocity that he achieved before the injury due to pain and numbness. It’s also called recurrent transient subluxation of the shoulder. (8)(6)<br>The dead arm syndrome is typically associated with anterior instability and a damaged anterior labrum, probably as a result of subluxation of the humeral head. This causes a transient stretch to the brachial plexus during a hard throw. (10)<br>The phenomenon is a disorder that can have different causes. Mostly it are problems of the rotator cuff or the labrum. Instability of the shoulder or posterior capsular contracture may be a reason for the development of the dead arm syndrome. In addition, it can also be caused by calcification in the ball and socket joint, bone spurs in the acromion, impingement of the shoulder ligaments, biceps tendonitis, micro-instability, internal impingement and SLAP lesion. Psychological factors can also cause this condition. This syndrome may also occur during throwing, repetitive forceful serving in tennis, or working with the arm in a strained position above shoulder. (8)(10)(6) <br>The symptoms can exacerbate by the loss of the posterior rollback. This leads to anterior translation and results in greater internal impingement posteriorly. (12)
<br>  


There are 2 categories of the dead arm syndrome: aware or unaware of subluxation.
<br>


3. Clinical Relevant Anatomy
== Differential Diagnosis&nbsp;  ==


4. Epidemiology/ etiology<br>The dead arm syndrome is seen most commonly in young athletes (21-30 years) or individuals whose arms have been powerful hyperextended in elevation and external rotation of the shoulder. (6)
It’s often misdiagnosed as other shoulder pathology or cervical lesion. There are some factors that differentiate the dead arm syndrome from the other causes of shoulder disability. First it usually appears by young athletic adults (21-30 years). It also has a characteristic history of forceful overextension of the shoulder and there is a positive apprehension test with relocation. <ref name="6a" />


It’s common for people who participate in repetitive throwing sports because the arm is repetitive being turned out backwards as far as possible (external rotation) to create potential energy in the wind up phase prior to the forward acceleration phase. It’s been postulated that the pain is due to an overstretched anterior capsule of the shoulder. Because of this, the ball of the shoulder can shift forward creating an impingement of structures lying in the front of the shoulder joint, which result in pain an the inability to throw. (8)
<br>  
 
5. Differential Diagnosis <br>It’s often misdiagnosed as other shoulder pathology or cervical lesion. There are some factors that differentiate the dead arm syndrome from the other causes of shoulder disability. First it usually appears by young athletic adults (21-30 years). It also has a characteristic history of forceful overextension of the shoulder and there is a positive apprehension test with relocation.(6)
 
6. Examination<br>There is a positive apprehension test. This test can be carried out when the patient is either in a standing or in a lying position. The shoulder is moved passively into maximum external rotation and in abduction. Then forward pressure is applied to the posterior aspect of the humeral head. The therapist give pressure against the caput humeri to anterior. The test is positive when the patient suddenly becomes apprehensive, complains of pain in the shoulder and has the feeling that the shoulder will come out of the joint considered a positive test. <br>In the absence of a strongly positive apprehension test, one should suspect that the shoulder disability is caused by something other than transient subluxation. (6)
 
Photo:&nbsp; test 1: http://www.fammed.wisc.edu/our-department/media/623/apprehension-relocation-test


<br>  
<br>  


<br>7. Medical management<br>Dead arm syndrome need to be treated. If there's a SLAP lesion, surgery is necessary to repair the problem. If the injury is incurred before a SLAP tear, then physical therapy with stretching and exercise can help to correct muscle imbalance and strengthen rotator cuff; if problems persist or recur, orthopaedic devices may be necessary. (7)
== Examination&nbsp; ==


Surgery when there is also a SLAP lesion: <br>The standard Bankart procedure is used when the capsule and labrum are avulsed from the glenoid rim. Three drill-holes are made in the anterior glenoid rim. 1 <br>Three sutures are used to reanach the lateral flap of the capsule to bone. 2<br>Once the sutures are tied, four of the six ends of the tied sutures are passed through the medial flap of the capsule as shown 3 <br>And they are tied to one another as indicated to form a double-layered reinforcement at the site of repair 4. (6)<br>&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; Photo A: standard Bankart (6)
There is a positive apprehension test. This test can be carried out when the patient is either in a standing or in a lying position. The shoulder is moved passively into maximum external rotation and in abduction. Then forward pressure is applied to the posterior aspect of the humeral head. The therapist give pressure against the caput humeri to anterior. The test is positive when the patient suddenly becomes apprehensive, complains of pain in the shoulder and has the feeling that the shoulder will come out of the joint considered a positive test. 
In the absence of a strongly positive apprehension test, one should suspect that the shoulder disability is caused by something other than transient subluxation. <ref name="6a" /><br>  


If a Bankart lesion is not present, a modified repair or capsulorrhaphy is performed. Since the labrum is still attached to the glenoid, it can be used to anchor the sutures . (6)
<br> {{#ev:youtube|qKqJRrms4u8}}<br>


Photo B: Bankart lesion not present (6)&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; <br>&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp; <br>8. Physical therapy management<br>Treatment includes physical therapy similar to that outlined for shoulder instability and labrum injuries. Surgery may be needed to correct the instability, as well as to repair injuries to the glenoid labrum. (10)<br>Once the inflammation and pain have resolved, the patient is subjected to a return to throw program. This takes about 4 weeks. (6)
<br>  


Return of full ROM and flexibility is needed before beginning strengthening exercises. These included resisted internal rotation, external rotation, and abduction of the shoulder to strengthen the muscles of the rotator cuff which stabilize the head of the humerus. This program, which is best carried out for three to four months, can decrease the pain and disability. (6) (6b) (10)
== Management / Intervention<br>  ==


Sometimes, it evolves into a full clinical picture of the posteo-superior impingement with a development of a SLAP lesion. Then there is need of a surgical treatment. (5)<br>Rehabilitation of athletes with the dead arm syndrome must include the entire kinetic chain. (6)
Treatment includes physical therapy similar to that outlined for shoulder instability and labrum injuries. Surgery may be needed to correct the instability, as well as to repair injuries to the glenoid labrum.<ref name="10" />&nbsp;
Once the inflammation and pain have resolved, the patient is subjected to a return to throw program. This takes about 4 weeks.<ref name="6a" />&nbsp;


<br>9. Key research<br>CR Rowe and B Zarins, Recurrent transient subluxation of the shoulder, J Bone Joint Surg Am. 1981;63:863-872. level of evidence: 2B
<br>Return of full ROM and flexibility is needed before beginning strengthening exercises. These included resisted internal rotation, external rotation, and abduction of the shoulder to strengthen the muscles of the rotator cuff which stabilize the head of the humerus. This program, which is best carried out for three to four months, can decrease the pain and disability.<ref name="6a" /><ref name="6b">Ho CY, The effectiveness of manual therapy in the management of musculoskeletal disorders of the shoulder: a systematic review, Man Ther. 2009 Oct;14(5):463-74. doi: 10.1016/j.math.2009.03.008. Epub 2009 May 21.</ref><ref name="10" />&nbsp;Rehabilitation of athletes with the dead arm syndrome must include the entire kinetic chain.<ref name="6a" />


10. References<br>Articles:<br>(1) Burkhart SS, Morgan CD, Kibler WB. The disabled throwing shoulder: spectrum of pathology Part I: pathoanatomy and biomechanics. Arthroscopy 2003; 25(4):945-949
<br>Sometimes, it evolves into a full clinical picture of the posteo-superior impingement with a development of a SLAP lesion. Then there is need of a surgical treatment.<ref name="5">Kibler WB. The role of the scapula in athletic shoulder function. Am J Sports Med 1998;26:325-337</ref>&nbsp;
SLAP lesions are repaired through arthroscopy, there are different types of SLAP lesions and the type would determine the repair option.<ref>http://orthoinfo.aaos.org/topic.cfm?topic=A00627</ref>&nbsp;&nbsp;


(2) O’Brien SJ. The trans-rotator cuff approach to SLAP lesions. Technical aspects for repair and a clinical follow-up of 31 patients at a minimum of 2 years. Arthroscopy 2002;18:372-377.
<br>


(3) Pagnini MJ. Arthroscopic fixation of superior labral lesions using a biodegradable implant. A preliminary report. Arthroscopy 1995;11:194-198.
<br>


(4) Warner JJP. Arthroscopic fixation of combined Bankart superior labral anterior posterior lesions. Techniques and preliminary results. Arthroscopy 1994;10:383-391
== Resources <br>  ==


(5) Kibler WB. The role of the scapula in athletic shoulder function. Am J Sports Med 1998;26:325-337
*http://www.uwhealth.org/files/uwhealth/docs/pdf/sm14888_slap_repair6.pdf
*http://fysiovaardig.boom.nl/?id=bju240407.30072008132020
*Burkhart SS, Morgan CD, Kibler WB. The disabled throwing shoulder: spectrum of pathology Part I: pathoanatomy and biomechanics. Arthroscopy 2003; 25(4):945-949
*O’Brien SJ. The trans-rotator cuff approach to SLAP lesions. Technical aspects for repair and a clinical follow-up of 31 patients at a minimum of 2 years. Arthroscopy 2002;18:372-377.
*Pagnini MJ. Arthroscopic fixation of superior labral lesions using a biodegradable implant. A preliminary report. Arthroscopy 1995;11:194-198.
*Warner JJP. Arthroscopic fixation of combined Bankart superior labral anterior posterior lesions. Techniques and preliminary results. Arthroscopy 1994;10:383-391
*Ralph M. Buschbacher,Nathan D. Prahlow, M.D.,Shashank J. Dave, Sports Medicine &amp; Rehabilitation, 2009, p 59<br>
*http://hss.edu/onthemove/dead-arm-syndrome-in-tennis-players/#.UYJwhmFCTwo


(6a) CR Rowe and B Zarins, Recurrent transient subluxation of the shoulder, J Bone Joint Surg Am. 1981;63:863-872. level of evidence: 2B
<br>


(6b) Ho CY, The effectiveness of manual therapy in the management of musculoskeletal disorders of the shoulder: a systematic review, Man Ther. 2009 Oct;14(5):463-74. doi: 10.1016/j.math.2009.03.008. Epub 2009 May 21.  
== Recent Related Research (from [http://www.ncbi.nlm.nih.gov/pubmed/ Pubmed]) ==
<div class="researchbox">
<rss>http://www.ncbi.nlm.nih.gov/entrez/eutils/erss.cgi?rss_guid=1XYaWIOtNnK9vlQcYPnBNZMyFM6RAujCCqwzHeBzxES-oJi12T|charset=UTF-8|short|max=10</rss>


Sites:<br>(7) http://medical-dictionary.thefreedictionary.com/  
<br>  
</div>
== References<br> ==


(8)&nbsp; http://www.coreconcepts.com.sg/mcr/the-disabled-throwing-shoulder-the-%E2%80%9Cdead-arm%E2%80%9D/
<references />.  


(9) http://hss.edu/onthemove/dead-arm-syndrome-in-tennis-players/#.UYJwhmFCTwo
<br>


Books:<br>(10) Richard B. Birrer,Bernard A.. Griesemer,Mary B. Cataletto, M.D.Pediatric sports medicine for primary care, 2002,&nbsp; p348 <br>&nbsp; <br>(11) Ralph M. Buschbacher,Nathan D. Prahlow, M.D.,Shashank J. Dave, Sports Medicine &amp; Rehabilitation, 2009, p 59
<br>  
 
(12) Donald H. Johnson, M.D, Practical orthopaedic sports medicine &amp; arthroscopy, 2007


Visual:<br>(13) http://fysiovaardig.boom.nl/?id=bju240407.30072008132020
[[Category:Condition]] [[Category:Sports_injuries]] [[Category:Shoulder]] [[Category:Musculoskeletal/Orthopaedics|Orthopaedics]]

Revision as of 03:32, 30 July 2014

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Definition/Description
[edit | edit source]


“Dead Arm” is characterized by a sudden sharp or ’paralyzing’ pain when the shoulder is moved forcibly into a position of maximum external rotation in elevation or is subjected to a direct blow. The patient is no longer able to perform a throwing movement with the control and the velocity that he achieved before the injury due to pain and numbness. It’s also called recurrent transient subluxation of the shoulder.[1]Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title 
The dead arm syndrome is typically associated with anterior instability and a damaged anterior labrum, probably as a result of subluxation of the humeral head. This causes a transient stretch to the brachial plexus during a hard throw.Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title 
The phenomenon is a disorder that can have different causes. Mostly it are problems of the rotator cuff or the labrum. Instability of the shoulder or posterior capsular contracture may be a reason for the development of the dead arm syndrome. In addition, it can also be caused by calcification in the ball and socket joint, bone spurs in the acromion, impingement of the shoulder ligaments, biceps tendonitis, micro-instability, internal impingement and SLAP lesion. Psychological factors can also cause this condition. This syndrome may also occur during throwing, repetitive forceful serving in tennis, or working with the arm in a strained position above shoulder.[1]Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive titleCite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title  
The symptoms can exacerbate by the loss of the posterior rollback. This leads to anterior translation and results in greater internal impingement posteriorly.Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title 


There are 2 categories of the dead arm syndrome: aware or unaware of subluxation.



Epidemiology / Etiology
[edit | edit source]

The dead arm syndrome is seen most commonly in young athletes (21-30 years) or individuals whose arms have been powerful hyperextended in elevation and external rotation of the shoulder. [1]  
It’s common for people who participate in repetitive throwing sports because the arm is repetitive being turned out backwards as far as possible (external rotation) to create potential energy in the wind up phase prior to the forward acceleration phase. It’s been postulated that the pain is due to an overstretched anterior capsule of the shoulder. Because of this, the ball of the shoulder can shift forward creating an impingement of structures lying in the front of the shoulder joint, which result in pain an the inability to throw. Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title  



Differential Diagnosis [edit | edit source]

It’s often misdiagnosed as other shoulder pathology or cervical lesion. There are some factors that differentiate the dead arm syndrome from the other causes of shoulder disability. First it usually appears by young athletic adults (21-30 years). It also has a characteristic history of forceful overextension of the shoulder and there is a positive apprehension test with relocation. [1]



Examination [edit | edit source]

There is a positive apprehension test. This test can be carried out when the patient is either in a standing or in a lying position. The shoulder is moved passively into maximum external rotation and in abduction. Then forward pressure is applied to the posterior aspect of the humeral head. The therapist give pressure against the caput humeri to anterior. The test is positive when the patient suddenly becomes apprehensive, complains of pain in the shoulder and has the feeling that the shoulder will come out of the joint considered a positive test. 
In the absence of a strongly positive apprehension test, one should suspect that the shoulder disability is caused by something other than transient subluxation. [1]




Management / Intervention
[edit | edit source]

Treatment includes physical therapy similar to that outlined for shoulder instability and labrum injuries. Surgery may be needed to correct the instability, as well as to repair injuries to the glenoid labrum.Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title 
Once the inflammation and pain have resolved, the patient is subjected to a return to throw program. This takes about 4 weeks.[1] 


Return of full ROM and flexibility is needed before beginning strengthening exercises. These included resisted internal rotation, external rotation, and abduction of the shoulder to strengthen the muscles of the rotator cuff which stabilize the head of the humerus. This program, which is best carried out for three to four months, can decrease the pain and disability.[1][2]Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title Rehabilitation of athletes with the dead arm syndrome must include the entire kinetic chain.[1]


Sometimes, it evolves into a full clinical picture of the posteo-superior impingement with a development of a SLAP lesion. Then there is need of a surgical treatment.Cite error: Invalid <ref> tag; name cannot be a simple integer. Use a descriptive title 
SLAP lesions are repaired through arthroscopy, there are different types of SLAP lesions and the type would determine the repair option.[3]  



Resources
[edit | edit source]


Recent Related Research (from Pubmed)[edit | edit source]

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References
[edit | edit source]

  1. ↑ 1.0 1.1 1.2 1.3 1.4 1.5 1.6 1.7 CR Rowe and B Zarins, Recurrent transient subluxation of the shoulder, J Bone Joint Surg Am. 1981;63:863-872. level of evidence: 2B
  2. ↑ Ho CY, The effectiveness of manual therapy in the management of musculoskeletal disorders of the shoulder: a systematic review, Man Ther. 2009 Oct;14(5):463-74. doi: 10.1016/j.math.2009.03.008. Epub 2009 May 21.
  3. ↑ http://orthoinfo.aaos.org/topic.cfm?topic=A00627

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