Friction Massage: Difference between revisions
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== Purpose<br> == | == Purpose<br> == | ||
The role of the therapist is to minimize the scarring that develops perpendicular to the actin and myosin filaments, to facilitate the proliferation of the fibroblasts in the scar and to develop a strong, mobile scar that won’t be a source of recidivate pain when returning to normal activities. | |||
The goal of friction massage is to influence cell behavior in all soft tissues. Friction massage is supposed to induce : | |||
• Traumatic hyperemia, which helps to evacuate pain triggering metabolites. | |||
• Movement of the affected structure which prevents or destroys adhesions and helps optimize the quality of scar tissue and mechanoreceptor stimulation. | |||
• Stimulation of mechanoreceptors, producing a quantity of afferent impulses that stimulate a temporary analgesia. | |||
• Fibroblastic proliferation, responsible for the repair en regeneration of collagen. | |||
• Realignment of collagen fibers, determined by the magnitude of applied pressure. | |||
== Technique<br> == | == Technique<br> == | ||
Revision as of 17:44, 30 December 2010
Original Editors - Laurence Leysen Lead Editors - Your name will be added here if you are a lead editor on this page. Read more.
Search Strategy[edit | edit source]
add text here related to databases searched, keywords, and search timeline
Definition/Description[edit | edit source]
Friction is defined as “an accurately delivered penetrating pressure applied through fingertips” . Cyriax, who is seen as the founder of the friction therapy, believed that deep frictions are appropriate for the treatment of tendinitis, tenosynovitis, muscle strains, ligament lesions and scar healing .
Clinically Relevant Anatomy[edit | edit source]
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Purpose
[edit | edit source]
The role of the therapist is to minimize the scarring that develops perpendicular to the actin and myosin filaments, to facilitate the proliferation of the fibroblasts in the scar and to develop a strong, mobile scar that won’t be a source of recidivate pain when returning to normal activities.
The goal of friction massage is to influence cell behavior in all soft tissues. Friction massage is supposed to induce :
• Traumatic hyperemia, which helps to evacuate pain triggering metabolites.
• Movement of the affected structure which prevents or destroys adhesions and helps optimize the quality of scar tissue and mechanoreceptor stimulation.
• Stimulation of mechanoreceptors, producing a quantity of afferent impulses that stimulate a temporary analgesia.
• Fibroblastic proliferation, responsible for the repair en regeneration of collagen.
• Realignment of collagen fibers, determined by the magnitude of applied pressure.
Technique
[edit | edit source]
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Key Research[edit | edit source]
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Resources
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Clinical Bottom Line[edit | edit source]
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Recent Related Research (from Pubmed)[edit | edit source]
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References[edit | edit source]
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