Hypermobile Meniscus: Difference between revisions
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== Mechanism of Injury / Pathological Process<br> == | == Mechanism of Injury / Pathological Process<br> == | ||
LMH is thought to be related to a congenital deficiency in the capsular attachments, or an atraumatic injury to the ligamentous attachments, specifically the PMF. Typically, hypermobility is due to an overuse injury, as well as atraumatic hypermobility. Studies have found in many cases of anterior cruciate ligament (ACL, and/or posterolateral injuries to the knee have concurrent damage to the PMF, which may result in LMH. | LMH is thought to be related to a congenital deficiency in the capsular attachments, or an atraumatic injury to the ligamentous attachments, specifically the PMF. Typically, hypermobility is due to an overuse injury, as well as atraumatic hypermobility. Studies have found in many cases of anterior cruciate ligament (ACL), and/or posterolateral injuries to the knee have concurrent damage to the PMF, which may result in LMH. | ||
An isolated incidence of LMH is rare. | An isolated incidence of LMH is rare. | ||
Revision as of 02:40, 21 March 2023
Top Contributors - Robert Pierce, Madisyn Melchor, Kim Jackson, Matt Huey, Cindy John-Chu and Briana Marin
Clinically Relevant Anatomy
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Lateral meniscus hypermobility (LMH) is excess motion at the lateral meniscus that can cause lateral knee pain or tenderness, locking of the knee, or limitation of the range of motion during knee flexion. It is a rare, uncommon condition.
The lateral meniscus is a C-shaped cartilaginous disc that sits on the convex, lateral tibial plateau. In a stable knee, the lateral meniscus is smaller, thinner, and more mobile than the medial meniscus. The menisci have various functions – load transmission, shock absorption, joint lubrication, nutrition, secondary mechanical stability, and guiding of movements.
Compared to the medial meniscus, the lateral meniscus has less stabilizers at its posterolateral aspect. The stabilizers of the lateral meniscus include the – popliteomeniscal fascicles (PMF), the posterior capsule, the meniscofemoral ligaments and the posterior meniscotibial ligament that are divided by popliteus tendon as it runs through the popliteal hiatus. These attachments to the posterior knee prevent the subluxation of the posterior horn of the lateral meniscus into the knee joint.
Mechanism of Injury / Pathological Process
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LMH is thought to be related to a congenital deficiency in the capsular attachments, or an atraumatic injury to the ligamentous attachments, specifically the PMF. Typically, hypermobility is due to an overuse injury, as well as atraumatic hypermobility. Studies have found in many cases of anterior cruciate ligament (ACL), and/or posterolateral injuries to the knee have concurrent damage to the PMF, which may result in LMH.
An isolated incidence of LMH is rare.
Clinical Presentation[edit | edit source]
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Diagnostic Procedures[edit | edit source]
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Outcome Measures[edit | edit source]
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Management / Interventions
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Differential Diagnosis
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- Bucket-handle Meniscal Lesion
- Articular Cartilage Lesion
- Anterior Cruciate Ligament (ACL) Tear
- Ligamentous Instability
- Patellar Pathology
- Patellar Dislocation
Resources
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