Theories of Pain: Difference between revisions

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This theory consider that peripheral sensory receptors, responding to touch, warmth and other non-damaging as well as to damaging stimuli, give rise to non-painful or painful experiences as a result of differences in the patterns [in time] of the signals sent through the nervous system.<ref name="site1" />  
This theory consider that peripheral sensory receptors, responding to touch, warmth and other non-damaging as well as to damaging stimuli, give rise to non-painful or painful experiences as a result of differences in the patterns [in time] of the signals sent through the nervous system.<ref name="site1" />  
Goldschneider (1920) proposed that there is no separate system for perceiving pain, and the receptors for pain are shared with other senses, such as of touch. According to this view, people feel pain when certain patterns of neural ctivity occur, such as when appropriate types of activity reach excessively high levels in the brain. These patterns occur only with intense stimulation. Because strong and mild stimuli of the same sense modality produce different patterns of neural activity, being hit hard feels painful, but being caressed does not.<ref name="site2" />


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== References  ==
== References  ==

Revision as of 20:47, 17 March 2014


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Introduction[edit | edit source]

Several theoretical frameworks have been proposed to explain the physiological basis of pain, although none yet completely accounts for all aspects of pain perception. A number of theories have been postulated to describe mechanisms underlying pain perception. These theories date back several centuries and even millennia (Kenins 1988; Perl 2007; Rey 1995)


1. Specificity Theory [edit | edit source]

This theory considers pain as an independent sensation with specialised peripheral sensory receptors [nociceptors], which respond to damage and send signals through pathways (along nerve fibres) in the nervous system to target centres in the brain. These brain centres process the signals to produce the experience of pain.[1]

Von Frey (1895) argued that the body has a separate sensory system for perceiving pain—just as it does for hearing and vision and this system contains its own special receptors for de:ecting pain stimuli, its own peripheral nerves and pathway to the brain, and its own area of the brain for processing pain signals. But this structure is not correct. [2]

2.  Pattern Theory[edit | edit source]

This theory consider that peripheral sensory receptors, responding to touch, warmth and other non-damaging as well as to damaging stimuli, give rise to non-painful or painful experiences as a result of differences in the patterns [in time] of the signals sent through the nervous system.[3]

Goldschneider (1920) proposed that there is no separate system for perceiving pain, and the receptors for pain are shared with other senses, such as of touch. According to this view, people feel pain when certain patterns of neural ctivity occur, such as when appropriate types of activity reach excessively high levels in the brain. These patterns occur only with intense stimulation. Because strong and mild stimuli of the same sense modality produce different patterns of neural activity, being hit hard feels painful, but being caressed does not.[2]




References[edit | edit source]