Assessment of Traumatic Brain Injury: Difference between revisions

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*and [[Stroke|page in Physiopedia related to the above example document]]
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Revision as of 11:01, 9 July 2019

Welcome to Traumatic Brain Injury Content Creation Project. This page is being developed by participants of a project to populate the Traumatic Brain Injury Section of Physiopedia. 
  • Please do not edit unless you are involved in this project, but please come back in the near future to check out new information!!  
  • If you would like to get involved in this project and earn accreditation for your contributions, [[[Special:Contact|please get in touch]]]!

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Preparation for Physiotherapy Assessment[edit | edit source]

Physiotherapy assessment commences once the patient is medically stable.

In view of the complexity of many patients with TBI, the assessment is frequently unable to be completed within a single session so is ongoing for the first few physiotherapy sessions. Close liaison with other members of the multidisciplinary team is extremely beneficial, and on occasion joint assessments by 2 or even 3 therapists from different disciplines can be beneficial.

Handling during Ax[edit | edit source]

In the initial stages following TBI, careful handling is essential when assessing patients, especially if they are in a minimally conscious state.

Information required before starting the assessment[edit | edit source]

The following information should be provided by the medical team before beginning the physiotherapy assessment:

  • State of consciousness of the patient - for further information see the Coma Recovery Scale page
  • Areas of the brain injured, including diffuse and secondary injury
  • Site of Decompression Craniotomy, if this has been performed on the patient[1]
  • Any other injuries sustained - patients who have TBI from road traffic accidents frequently also have a range of musculoskeletal, abdominal and chest injuries
  • Any impact on cognition of the patient - neuropsychological assessment is indicated

Areas of Assessment[edit | edit source]

Abnormal Muscle Tone[edit | edit source]

The major abnormalities in muscle tone encountered in this popularion are hypertonicity and spasticity.

It is noticeable that, in contrast to Stroke when there is often a period of low tone before high tone develops, in cases of severe TBI hypertonicity and spasticity can develop very quickly, sometimes as early as one week post TBI[2][3]. The symptoms may start to occur as sedation is reduced, or as the patient emerges from coma. A recent study concludes that "Signs of spasticity can often be noted within the first 4 weeks after brain injury and is more common in the upper than lower extremity. Impaired sensorimotor function is a predictor"[2]

Approximately 18% to 30% of all TBI patients suffer from spasticity that requires treatment[4]. Studies show that in the region of 85% of people with severe TBI demonstrate significant spasticity at a level which induces contracture[5][6].

Muscle Paresis[edit | edit source]

Muscle paresis very common following TBI[7]

Movement Disorders[edit | edit source]

Ataxia, dyspraxia, dyskinesia, or reduced motor control

Balance and vestibular dysfunction[edit | edit source]

This is very common in TBI, and varies enormously in degree[8]

Secondary Effects[edit | edit source]

  • Reduced joint and muscle range of movement occur as a result of a combination of factors, including prolonged bed-rest, hypertonicity and spasticity, and in some cases as a result of musculoskeletal injuries sustained in the accident
  • Pain may occur as a result of spasticity and hypertonicity

References[edit | edit source]

  1. Bohman LE, Schuster JM. Decompressive craniectomy for management of traumatic brain injury: an update. Curr Neurol Neurosci Rep. Nov 2013;13(11):392
  2. 2.0 2.1 Sunnerhagen KS, Opheim A, Alt Murphy M.Onset, Time course and prediction of spasticity after stroke or traumatic brain injury. Ann Phys Rehabil Med. 2018 May 16. pii: S1877-0657(18)30059-9. doi: 10.1016/j.rehab.2018.04.004. [Epub ahead of print]
  3. Prodip Bose, Jiamei Hou, and Floyd J Thompson Traumatic Brain Injury (TBI)-Induced Spasticity Chapter 14 Neurobiology, Treatment, and Rehabilitation
  4. Verplancke, D, Snape, S, Salisbury, CF et al. 2005. A randomised controlled trial of botulinum toxin on lower limb spasticity following acute acquired severe brain injury. Rehabil. 19, 117-125
  5. Corrigan J.D, Selassie A.W, Orman J.A. The epidemiology of traumatic brain injury. J Head Trauma Rehabil. 2010;25:72–80
  6. McGuire L. The epidemiology of traumatic brain injury, National Centers for Disease Control and Prevention. 2011.
  7. Ivanhoe, CB, Reistetter, TA, 2004. Spasticity: the misunderstood part of the upper motor neuron syndrome. Am. J. Phy. Med. Rehabil. 83, S3-S9
  8. Kleffelgaard I, Soberg H, Bruusgaard K, Tamber A, Langhammer B. Vestibular Rehabilitation After Traumatic Brain Injury: Case Series. Physical Therapy. 2015